Epstein-Barr virus (EBV)-DNA quantification in pediatric allogenic stem cell recipients: prediction of EBV-associated lymphoproliferative disease.
نویسندگان
چکیده
1. Rathbun RK, Christianson TA, Faulkner GR, et al. Interferon-g-induced apoptotic responses of Fanconi anemia group C hematopoietic progenitor cells involve caspase 8-dependent activation of caspase 3 family members. Blood. 2000;96:4204-4211. 2. Rathbun RK, Faulkner GR, Ostroski MH, et al. Inactivation of the Fanconi anemia group C (FAC) gene augments interferon-gamma-induced apoptotic responses in hematopoietic cells. Blood. 1997;90:974-985. 3. Fagerlie SR, Diaz J, Christianson TA, et al. Functional correction of FA-C cells with FANCC suppresses the expression of interferon gamma-inducible genes. Blood. 2001;97:3017-3024. 4. Koh PS, Hughes GC, Faulkner GR, Keeble WW, Bagby GC. The Fanconi anemia group C gene product modulates apoptotic responses to tumor necrosis factor-a and Fas ligand but does not suppress expression of receptors of the tumor necrosis factor receptor superfamily. Exp Hematol. 1999;27:1-8.
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Seropidemiology of Epstein – Barr virus infection among asymptomatic
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متن کاملEpstein-Barr virus (EBV)–DNA quantification in pediatric allogeneic stem cell recipients: prediction of EBV-associated lymphoproliferative disease
1. Rathbun RK, Christianson TA, Faulkner GR, et al. Interferon-g-induced apoptotic responses of Fanconi anemia group C hematopoietic progenitor cells involve caspase 8-dependent activation of caspase 3 family members. Blood. 2000;96:4204-4211. 2. Rathbun RK, Faulkner GR, Ostroski MH, et al. Inactivation of the Fanconi anemia group C (FAC) gene augments interferon-gamma-induced apoptotic respons...
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عنوان ژورنال:
- Blood
دوره 99 7 شماره
صفحات -
تاریخ انتشار 2002